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Ayurveda Dharma is distributed copyleft via this GNU Free Documentation License (FDL) and/or under Creative Commons License Attribution-Share Alike 3.0 United States: Since words create our reality (see prajna), there are certain words to avoid: Please see also: Words to Avoid

For osteoarthritis: Take 1 tablet of Yogaraj Guggulu twice a day. At night, take one half teaspoon Gandharva Haritaki (Haritaki sauteed in Castor oil) with warm water. If you don't have Gandharva Haritaki, use Ginger tea with Castor oil, as described above.

Fair Use Source: Vasant Lad, Complete Book of Ayurvedic Home Remedies, Three Rivers Press, 1999, ISBN 0609802860, p. 131

Osteoarthritis From Wikipedia, the free encyclopedia Jump to: navigation, search Osteoarthritis Classification and external resources ICD-10 M15.-M19., M47. ICD-9 715 OMIM 165720 DiseasesDB 9313 MedlinePlus 000423 eMedicine med/1682 orthoped/427 pmr/93 radio/492 MeSH D010003

Osteoarthritis (OA, also known as degenerative arthritis, degenerative joint disease), is a clinical syndrome in which low-grade inflammation results in pain in the joints, caused by abnormal wearing of the cartilage that covers and acts as a cushion inside joints and destruction or decrease of synovial fluid that lubricates those joints. As the bone surfaces become less well protected by cartilage, sub-chondral bone can eventually be exposed leading to a proliferation of ivory-like, dense, reactive bone in central areas of cartilage loss, a process called eburnation.[1] The patient increasingly experiences pain upon weight bearing, including walking and standing. Due to decreased movement because of the pain, regional muscles may atrophy, and ligaments may become more lax.[2] OA is the most common form of arthritis,[2] and the leading cause of chronic disability in the United States.[3]

“Osteoarthritis” is derived from the Greek word “osteo”, meaning “of the bone”, “arthro”, meaning “joint”, and “itis”, meaning inflammation, although many sufferers have little or no inflammation. A common misconception is that OA is due solely to wear and tear, since OA typically is not present in younger people. However, while age is correlated with OA incidence, this correlation merely illustrates that OA is a process that takes time to develop. There is usually an underlying cause for OA, in which case it is described as secondary OA. If no underlying cause can be identified it is described as primary OA. “Degenerative arthritis” is often used as a synonym for OA, but the latter involves both degenerative and regenerative changes.

OA affects nearly 21 million people in the United States, accounting for 25% of visits to primary care physicians, and half of all NSAID (Non-Steroidal Anti-Inflammatory Drugs) prescriptions. It is estimated that 80% of the population will have radiographic evidence of OA by age 65, although only 60% of those will show symptoms.[4] In the United States, hospitalizations for osteoarthritis soared from about 322,000 in 1993 to 735,000 in 2006.[5]

Signs and symptoms

The main symptom is acute pain, causing loss of ability and often stiffness. “Pain” is generally described as a sharp ache, or a burning sensation in the associated muscles and tendons. OA can cause a crackling noise (called “crepitus”) when the affected joint is moved or touched, and patients may experience muscle spasm and contractions in the tendons. Occasionally, the joints may also be filled with fluid. Humid and cold weather increases the pain in many patients.[6][7]

OA commonly affects the hips, feet, spine, and the large weight bearing joints, such as the hips and knees, although in theory, any joint in the body can be affected. As OA progresses, the affected joints appear larger, are stiff and painful, and usually feel worse, the more they are used throughout the day, thus distinguishing it from rheumatoid arthritis. Heberden's nodes may form in osteoarthritis

In smaller joints, such as at the fingers, hard bony enlargements, called Heberden's nodes (on the distal interphalangeal joints) and/or Bouchard's nodes (on the proximal interphalangeal joints), may form, and though they are not necessarily painful, they do limit the movement of the fingers significantly. OA at the toes leads to the formation of bunions, rendering them red or swollen.

OA is the most common cause of water on the knee, an accumulation of excess fluid in or around the knee joint. [8]

[edit] Causes

Although it commonly arises from trauma, osteoarthritis often affects multiple members of the same family, suggesting that there is hereditary susceptibility to this condition. A number of studies have shown that there is a greater prevalence of the disease between siblings and especially identical twins, indicating a hereditary basis [9]. Up to 60% of OA cases are thought to result from genetic factors. Researchers are also investigating the possibility of allergies, infections, or fungi as a cause.

[edit] Two types

OA affects nearly 21 million people in the United States, accounting for 25% of visits to primary care physicians, and half of all NSAID (Non-Steroidal Anti-Inflammatory Drugs) prescriptions. It is estimated that 80% of the population will have radiographic evidence of OA by age 65, although only 60% of those will be symptomatic.[4] Treatment is with NSAIDs, local injections of glucocorticoid or hyaluronan, and in severe cases, with joint replacement surgery. There has been no cure for OA, as cartilage has not been induced to regenerate. However, if OA is caused by cartilage damage (for example as a result of an injury) Autologous Chondrocyte Implantation may be a possible treatment.[10] Clinical trials employing tissue-engineering methods have demonstrated regeneration of cartilage in damaged knees, including those that had progressed to osteoarthritis.[11] Further, in January 2007, Johns Hopkins University was offering to license a technology of this kind, [12] listing several clinical competitors in its market analysis.

[edit] Primary Primary OA in the left knee of an elderly female.

This type of OA is a chronic degenerative disorder related to but not caused by aging, as there are people well into their nineties who have no clinical or functional signs of the disease. As a person ages, the water content of the cartilage decreases due to a reduced proteoglycan content, thus causing the cartilage to be less resilient. Without the protective effects of the proteoglycans, the collagen fibers of the cartilage can become susceptible to degradation and thus exacerbate the degeneration. Inflammation of the surrounding joint capsule can also occur, though often mild (compared to that which occurs in rheumatoid arthritis). This can happen as breakdown products from the cartilage are released into the synovial space, and the cells lining the joint attempt to remove them. New bone outgrowths, called “spurs” or osteophytes, can form on the margins of the joints, possibly in an attempt to improve the congruence of the articular cartilage surfaces. These bone changes, together with the inflammation, can be both painful and debilitating.

[edit] Secondary

This type of OA is caused by other factors but the resulting pathology is the same as for primary OA:

   * Congenital disorders, such as:
         o Congenital hip luxation
         o People with abnormally-formed joints (e.g. hip dysplasia (human)) are more vulnerable to OA, as added stress is specifically placed on the joints whenever they move. [However, recent studies have shown that double-jointedness may actually protect the fingers and hand from osteoarthritis.]
   * Cracking joints—the evidence is weak at best that this has any connection to arthritis.
   * Diabetes.
   * Inflammatory diseases (such as Perthes' disease), (Lyme disease), and all chronic forms of arthritis (e.g. costochondritis, gout, and rheumatoid arthritis). In gout, uric acid crystals cause the cartilage to degenerate at a faster pace.
   * Injury to joints, as a result of an accident.
   * A joint infection, e.g. from an injury.
   * Hormonal disorders.
   * Ligamentous deterioration or instability may be a factor.
   * Obesity. Obesity puts added weight on the joints, especially the knees.
   * Sports injuries, or similar injuries from exercise or work. Certain sports, such as running or football, put undue pressure on the knee joints. Injuries resulting in broken ligaments can lead to instability of the joint and over time to wear on the cartilage and eventually osteoarthritis.
   * Pregnancy
   * Alkaptonuria
   * Hemochromatosis and Wilson's disease

[edit] Diagnosis

Diagnosis is normally done through x-rays. This is possible because loss of cartilage, subchondral (“below cartilage”) sclerosis, subchondral cysts from synovial fluid entering small microfractures under pressure, narrowing of the joint space between the articulating bones, and bone spur formation (osteophytes) - from increased bone turnover in this inflammatory condition, show up clearly on x-rays. Plain films, however, often do not correlate well with the findings of physical examination of the affected joints.

With or without other techniques, such as MRI (magnetic resonance imaging), arthrocentesis and arthroscopy, diagnosis can be made by a careful study of the duration, location, the character of the joint symptoms, and the appearance of the joints themselves. As yet, there are no methods available to detect OA in its early and potentially treatable stages.

In 1990, the College of Rheumatology, using data from a multi-center study, developed a set of criteria for the diagnosis of hand osteoarthritis based on hard tissue enlargement and swelling of certain joints. These criteria were found to be 92% sensitive and 98% specific for hand osteoarthritis versus other entities such as rheumatoid arthritis and spondyloarthropities [13].

Related pathologies whose names may be confused with osteoarthritis include pseudo-arthrosis. This is derived from the Greek words pseudo, meaning “false”, and arthrosis, meaning “joint.” Radiographic diagnosis results in diagnosis of a fracture within a joint, which is not to be confused with osteoarthritis which is a degenerative pathology affecting a high incidence of distal phalangeal joints of female patients.

[edit] Treatment

Generally speaking, the process of clinically detectable osteoarthritis is irreversible, and typical treatment consists of medication or other interventions that can reduce the pain of OA and thereby improve the function of the joint.

[edit] Conservative care

No matter the severity or location of OA, conservative measures such as weight control, appropriate rest and exercise, and the use of mechanical support devices are usually beneficial. In OA of the knees, knee braces, a cane, or a walker can be helpful for walking and support. Regular exercise, if possible, in the form of walking or swimming,and not giving strong impacts is encouraged. Applying local heat before, and cold packs after exercise, can help relieve pain and inflammation, as can relaxation techniques. Heat — often moist heat — eases inflammation and swelling, and may improve circulation, which has a healing effect on the local area. Weight loss can relieve joint stress and may delay progression (Prevention suggestion cited here)[citation needed]. Proper advice and guidance by a health care provider is important in OA management, enabling people with this condition to improve their quality of life.

In 2002, a randomized, blinded assessor trial was published showing a positive effect on hand function with patients who practiced home joint protection exercises (JPE). Grip strength, the primary outcome parameter, increased by 25% in the exercise group versus no improvement in the control group. Global hand function improved by 65% for those undertaking JPE. [14]

[edit] Medical treatment

Medical treatment includes NSAIDs, local injections of glucocorticoid or hyaluronan, and in severe cases, with joint replacement surgery. There has been no cure for OA, as cartilage has not been induced to regenerate. However, if OA is caused by cartilage damage (for example as a result of an injury) Autologous Chondrocyte Implantation may be a possible treatment.[10] Clinical trials employing tissue-engineering methods have demonstrated regeneration of cartilage in damaged knees, including those that had progressed to osteoarthritis.[11] Further, in January 2007, Johns Hopkins University was offering to license a technology of this kind, [12] listing several clinical competitors in its market analysis.

[edit] Dietary

Supplements which may be useful for treating OA include:

[edit] Glucosamine

There is still controversy about glucosamine's effectiveness for OA of the knee.[15] A 2005 review concluded that glucosamine may improve symptoms of OA and delay its progression.[16] However, a subsequent large study suggests that glucosamine is not effective in treating OA of the knee[17], and a 2007 meta-analysis that included this trial states that glucosamine hydrochloride is not effective.[18]

[edit] Chondroitin

Along with glucosamine, chondroitin sulfate has become a widely used dietary supplement for treatment of osteoarthritis. A meta-analysis of randomized controlled trials found no benefit from chondroitin.[19] However, the Osteoarthritis Research Society International is in support of the use of chondroitin sulfate for OA.[citation needed]

[edit] Other supplements

   * Omega-3 fatty acid,a vitamin supplement comprised of important oils derived from fish.[citation needed]
   * Frankincense resin from trees in the genus Boswellia. In Ayurvedic medicine, Indian frankincense (Boswellia serrata) has been used for hundreds of years for treating arthritis.[20]
   * Bromelain, protease enzymes extracted from the plant family Bromeliaceae (pineapple), blocks some proinflammatory metabolites.[21]
   * Antioxidants, including vitamins C and E in both foods and supplements, provide pain relief from OA.[22]
   * Hydrolyzed collagen (hydrolysate) (a gelatin product) may also prove beneficial in the relief of OA symptoms, as substantiated in a German study by Beuker F. et al. and Seeligmuller et al. In their 6-month placebo-controlled study of 100 elderly patients, the verum group showed significant improvement in joint mobility.[citation needed]
   * Ginger (rhizome) extract - has improved knee symptoms moderately.[23]
   * Selenium deficiency has been correlated with a higher risk and severity of OA.[24]
   * Vitamin B9 (folate) and B12 (cobalamin) taken in large doses has been thought to reduce OA hand pain in one very small, non-quantitative study of 25 people. The results of which are extremely vague at best.[25] The risk from large doses would suggest that this is not a safe treatment.
   * Vitamin D deficiency has been reported in patients with OA, and supplementation with Vitamin D3 is recommended for pain relief.[26]
   * Bone Morphogenetic Protein 6 (BMP-6) has recently been shown to have a functional role in the maintenance of joint integrity and is now being produced in an orally ingested form. [27]

Other nutritional changes shown to aid in the treatment of OA include decreasing saturated fat intake[28] and using a low energy diet to decrease body fat.[29] Lifestyle change may be needed for effective symptomatic relief, especially for knee OA.[30]

[edit] Complications

Dealing with chronic pain can be difficult and result in depression. Communicating with other patients and caregivers can be helpful, as can maintaining a positive attitude. People who take control of their treatment, communicate with their health care provider, and actively manage their arthritis experience can reduce pain and improve function.[citation needed]

[edit] Specific medications

[edit] Paracetamol

A mild pain reliever may be sufficiently efficacious. Paracetamol (tylenol/acetaminophen), is commonly used to treat the pain from OA, although unlike NSAIDs, acetaminophen does not treat the inflammation.[citation needed] A randomized controlled trial comparing paracetamol with ibuprofen in x-ray-proven mild to moderate osteoarthritis of the hip or knee found equal benefit.[31] However, acetaminophen at a dose of 4 grams per day can increase liver function tests.[32]

[edit] Non-steroidal anti-inflammatory drugs

In more severe cases, non-steroidal anti-inflammatory drugs (NSAID) may reduce both the pain and inflammation; they all act by inhibiting the formation of prostaglandins, which play a central role in inflammation and pain. Most prominent drugs in the class include diclofenac, ibuprofen, naproxen and ketoprofen. High oral drug doses are often required. However, diclofenac has been found to cause damage to the articular cartilage. Even more importantly all systemic NSAIDs are rather taxing on the gastrointestinal tract, and may cause stomach upset, cramping, diarrhea, and peptic ulcer. Such systemic adverse side effects are normally not observed when using NSAIDs topically, that is, on the skin around the target area. The typically weak and/or short-lived therapeutic effect of such topical treatments may be improved by using the drug in more modern formulations, including or ketoprofen associated with the Transfersome carriers or diclofenac in DMSO solution.

[edit] COX-2 selective inhibitors

Another type of NSAID, COX-2 selective inhibitors (such as celecoxib, and the withdrawn rofecoxib and valdecoxib) reduce this risk substantially. These latter NSAIDs carry an elevated risk for cardiovascular disease, and some have now been withdrawn from the market.

[edit] Corticosteroids

Most doctors nowadays avoid the use of steroids in the treatment of OA as their effect is modest and the adverse effects may outweigh the benefits.

[edit] Narcotics

For moderate to severe pain, narcotic pain relievers such as tramadol, and eventually opioids (hydrocodone, oxycodone or morphine) may be necessary.

[edit] Topical

“Topical treatments” are treatments designed for local application and action. There are several NSAIDs available for topical use (e.g. diclofenac, ibuprofen, and ketoprofen) with little, if any, systemic side-effects and at least some therapeutic effect. The more modern NSAID formulations for direct use, containing the drugs in an organic solution or the Transfersome carrier based gel, reportedly, are as effective as oral NSAIDs.

Creams and lotions, containing capsaicin, are effective in treating pain associated with OA if they are applied with sufficient frequency.

Severe pain in specific joints can be treated with local lidocaine injections or similar local anaesthetics, and glucocorticoids (such as hydrocortisone). Corticosteroids (cortisone and similar agents) may temporarily reduce the pain. Certain anti-inflammatory medications, such as dexamethasone, can also be used in a procedure called iontophoresis, which uses mild electrical current to transfer the medication through the skin.

[edit] Surgery

If the above management is ineffective, joint replacement surgery may be required. Individuals with very painful OA joints may require surgery such as fragment removal, repositioning bones, or fusing bone to increase stability and reduce pain. Surgical intervention for osteoarthritis of the knee may be no better than placebo at relieving symptoms.[33]

[edit] Acupuncture

A meta-analysis of randomized controlled trials of acupuncture for knee osteoarthritis concluded “clinically relevant benefits, some of which may be due to placebo or expectation effects”.[34]

[edit] Prognosis

The most common course of OA is an intermittent, progressive worsening of symptoms over time, although in some patients the disease stabilizes. Prognosis also varies depending on which joint is involved.

Factors associated with progression of OA:

   * Knees: High body mass index, varus or valgus knee deformity.
   * Hips: Night pain, presence of femoral osteophytes, and subchondral sclerosis in females.
   * Hands: Older age.
   * Feet/Ankles

[edit] Additional images

Examples of damaged cartilage in gross pathological specimen from sows. (a) cartilage erosion (b) cartilage ulceration © cartilage repair (d) osteophyte (bone spur) formation.

[edit] See also

   * Articular cartilage repair
   * Autologous Chondrocyte Implantation
   * Back pain
   * Chronic pain
   * Osteoimmunology
   * Prolotherapy
   * Partial knee replacement
   * Arthritis Care

[edit] References

  1. ^ Siddiqui, Furqan (2008-9-12). "Osteoarthritis". Emedicine. Retrieved on 2009-01-27.
  2. ^ a b Conaghan, Phillip. "Osteoarthritis - National clinical guideline for care and management in adults" (PDF). Retrieved on 2008-04-29.
  3. ^ "Prevalence of disabilities and associated health conditions among adults—United States, 1999". MMWR Morb Mortal Wkly Rep. 50 (7): 120–5. February 2001. PMID 11393491. 
  4. ^ a b Green GA (2001). "Understanding NSAIDs: from aspirin to COX-2". Clin Cornerstone 3 (5): 50–60. PMID 11464731. 
  5. ^ Hospitalizations for Osteoarthritis Rising Sharply Newswise, Retrieved on September 4, 2008.
  6. ^ McAlindon, T., Formica, M., Schmid, C.H., & Fletcher, J. (2007). Changes in barometric pressure and ambient temperature influence osteoarthritis pain. The American Journal of Medicine, 120(5), 429-434.
  7. ^ MedlinePlus Encyclopedia Osteoarthritis
  8. ^ Water on the knee,
  9. ^ Valdes AM, Spector TD (August 2008). "The contribution of genes to osteoarthritis". Rheum Dis Clin North Am. 34 (3): 581–603. doi:10.1016/j.rdc.2008.04.008. PMID 18687274. 
 10. ^ a b Autologous Chondrocyte Implantation
 11. ^ a b Hollander AP, Dickinson SC, Sims TJ, et al (2006). "Maturation of tissue engineered cartilage implanted in injured and osteoarthritic human knees". Tissue Eng. 12 (7): 1787–98. doi:10.1089/ten.2006.12.1787. PMID 16889509. 
 12. ^ a b Repairing knee joints by growing new cartilage using an injectable hydrogel
 13. ^ Altman R, Alarcón G, Appelrouth D, et al (1990). "The American College of Rheumatology criteria for the classification and reporting of osteoarthritis of the hand". Arthritis Rheum. 33 (11): 1601–10. doi:10.1002/art.1780331101. PMID 2242058. 
 14. ^ Stamm TA, Machold KP, Smolen JS, et al (2002). "Joint protection and home hand exercises improve hand function in patients with hand osteoarthritis: a randomized controlled trial". Arthritis Rheum. 47 (1): 44–9. doi:10.1002/art1.10246. PMID 11932877. 
 15. ^ "The effects of Glucosamine Sulphate on OA of the knee joint".
 16. ^ Poolsup N, Suthisisang C, Channark P, Kittikulsuth W (2005). "Glucosamine long-term treatment and the progression of knee osteoarthritis: systematic review of randomized controlled trials". The Annals of pharmacotherapy 39 (6): 1080–7. doi:10.1345/aph.1E576. PMID 15855241. 
 17. ^ McAlindon T, Formica M, LaValley M, Lehmer M, Kabbara K (November 2004). "Effectiveness of glucosamine for symptoms of knee osteoarthritis: results from an internet-based randomized double-blind controlled trial". Am J Med 117 (9): 643–9. doi:10.1016/j.amjmed.2004.06.023. PMID 15501201. 
 18. ^ Vlad SC, Lavalley MP, McAlindon TE, Felson DT (2007). "Glucosamine for pain in osteoarthritis: Why do trial results differ?". Arthritis & Rheumatism 56 (7): 2267–77. doi:10.1002/art.22728. PMID 17599746. 
 19. ^ Reichenbach S, Sterchi R, Scherer M, et al (2007). "Meta-analysis: chondroitin for osteoarthritis of the knee or hip". Ann. Intern. Med. 146 (8): 580–90. PMID 17438317. 
 20. ^ "JOINT RELIEF". Retrieved on 2009-01-12.
 21. ^ Brien S, Lewith G, Walker A (2004). "Bromelain as a Treatment for Osteoarthritis: a Review of Clinical Studies". Evidence-based complementary and alternative medicine: eCAM. 1 (3): 251–257. doi:10.1093/ecam/neh035. PMID 15841258. 
 22. ^ McAlindon TE, Jacques P, Zhang Y, et al (1996). "Do antioxidant micronutrients protect against the development and progression of knee osteoarthritis?". Arthritis Rheum. 39 (4): 648–56. PMID 8630116. 
 23. ^ Altman RD, Marcussen KC (2001). "Effects of a ginger extract on knee pain in patients with osteoarthritis". Arthritis Rheum. 44 (11): 2531–8. PMID 11710709. 
 24. ^ "UNC News release -- Study links low selenium levels with higher risk of osteoarthritis". Retrieved on 2007-06-22.
 25. ^ Flynn MA, Irvin W, Krause G (1994). "The effect of folate and cobalamin on osteoarthritic hands". J Am Coll Nutr 13 (4): 351–6. PMID 7963140. 
 26. ^ Arabelovic S, McAlindon TE (2005). "Considerations in the treatment of early osteoarthritis". Curr Rheumatol Rep 7 (1): 29–35. PMID 15760578. 
 27. ^ Bobacz K, Gruber R, Soleiman A, Erlacher L, Smolen JS, Graninger WB (2003). "Expression of bone morphogenetic protein 6 in healthy and osteoarthritic human articular chondrocytes and stimulation of matrix synthesis in vitro". Arthritis Rheum. 48 (9): 2501–8. doi:10.1002/art.11248. PMID 13130469. 
 28. ^ Wilhelmi G. Z Rheumatol. 1993 May-Jun; 52(3):174-9. Vasishta VG et al, Rotational Field Magnetic Resonance (RFQMR) in treatment of osteoarthritis of the knee joint, Indian Journal of Aerospace Medicine, 48 (2), 2004; 1-7.
 29. ^ Christensen R, Astrup A, Bliddal H (2005). "Weight loss: the treatment of choice for knee osteoarthritis? A randomized trial". Osteoarthr. Cartil. 13 (1): 20–7. doi:10.1016/j.joca.2004.10.008. PMID 15639633. 
 30. ^ De Filippis L, Gulli S, Caliri A, et al (2004). "[Epidemiology and risk factors in osteoarthritis: literature review data from "OASIS" study]" (in Italian) (PDF). Reumatismo 56 (3): 169–84. PMID 15470523. 
 31. ^ Bradley JD, Brandt KD, Katz BP, Kalasinski LA, Ryan SI (1991). "Comparison of an antiinflammatory dose of ibuprofen, an analgesic dose of ibuprofen, and paracetamol in the treatment of patients with osteoarthritis of the knee". N. Engl. J. Med. 325 (2): 87–91. PMID 2052056. 
 32. ^ Watkins PB, Kaplowitz N, Slattery JT, et al (2006). "Aminotransferase elevations in healthy adults receiving 4 grams of acetaminophen daily: a randomized controlled trial". JAMA 296 (1): 87–93. doi:10.1001/jama.296.1.87. PMID 16820551. 
 33. ^ Moseley JB, O'Malley K, Petersen NJ, et al (July 2002). "A controlled trial of arthroscopic surgery for osteoarthritis of the knee". N. Engl. J. Med. 347 (2): 81–8. doi:10.1056/NEJMoa013259. PMID 12110735. 
 34. ^ Manheimer E, Linde K, Lao L, Bouter LM, Berman BM (2007). "Meta-analysis: acupuncture for osteoarthritis of the knee". Ann. Intern. Med. 146 (12): 868–77. doi:10.1001/archinte.146.5.868. PMID 17577006. 

[edit] External links

   * American College of Rheumatology Factsheet on OA
   * Osteoarthritis The Arthritis Foundation
   * Arthritis Care major UK charity
   * WebMDHealth: Osteoarthritis Basics at WebMD
   * MedlinePlus: Osteoarthritis at National Institutes of Health
   * Osteoarthritis Clinical Trials Resource at
   * Overview at University of Maryland
   * Focuses on living with arthritis with links to support groups in 16 different countries at
   * BBC Coverage of Autologous Chondrocyte graft in UK
   * UK Health Charity covers Autologous Chondrocyte grafts
   * Treatment Information for Arthritis Sufferers

[hide] v • d • e Musculoskeletal disorders: Arthropathies (M00-M25, 710-719) Arthritis (monoarthritis/ polyarthritis) Septic arthritis

Seronegative spondyloarthropathy: Reactive arthritis · Psoriatic arthritis

Rheumatoid arthritis: Juvenile idiopathic arthritis · Adult-onset Still's disease · Felty syndrome

Crystal arthropathy: Gout · Chondrocalcinosis

Osteoarthritis: Heberden's node · Bouchard's nodes Palindromic rheumatism Specific joints Upper limb

shoulder (Winged scapula, Adhesive capsulitis, Rotator cuff tear, Subacromial bursitis) · elbow (Cubitus valgus, Cubitus varus) · hand (Wrist drop, Boutonniere deformity, Swan neck deformity) Lower limb

hip (Protrusio acetabuli, Coxa valga, Coxa vara) · leg (Unequal leg length) · patella (Luxating patella, Chondromalacia patellae) · foot (Bunion/hallux valgus, Hallux varus, Hallux rigidus, Hammer toe, Foot drop, Flat feet, Club foot) General terms

Valgus deformity · Varus deformity Synovium and tendon Synovitis/Tenosynovitis (Calcific tendinitis, Stenosing tenosynovitis, Trigger finger, DeQuervain's syndrome) · Irritable hip · Ganglion cyst Bursa Bursitis (Olecranon, Prepatellar, Trochanteric, Subacromial) · Baker's cyst Other Hemarthrosis · Arthralgia · Osteophyte · Hypermobility see also congenital Retrieved from “” Categories: Arthritis | General practice


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Fundamentals of Ayurveda | Three Doshas | Vata | Pitta | Kapha

Ayurvedic Constitutions | Vata-Pitta | Pitta-Kapha | Kapha-Vata

Balancing Vata | Balancing Pitta | Balancing Kapha

Vata Diet | Pitta Diet Kapha Diet

Vata-Pitta Diet | Pitta-Kapha Diet | Tridoshic Diet

Ten Pairs of Attributes | Five Elements

Six Tastes | Ama | Agni | Ojas | Tejas | Prana

Organs and Ayurveda | Dhatus

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Ayurvedic Herbs | Top Ten Ayurvedic Herbs | Ayurvedic Material Medica

Amalaki | Shilajit | Guggulu | Ashwagandha | Brahmi | Musta

Organic Ayurveda | Ayurvedic Herb Suppliers

Growth - Manufacture - Quality | Heavy Metals in Ayurveda | Safety and Regulation

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Herbal Actions and Interactions | Language of Herbs | Ayurvedic Sanskrit

Herbal Traditions: Traditional Chinese Medicine

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Ayurvedic Food Therapy compared to Chinese | Energies of Foods

Environmental Toxins | Toxins and Ayurveda - Ama

Ayurvedic Digestion - Agni

Constipation and Ayurveda - Triphala

Irritable Bowel Syndrome and Ayurveda IBS - Avipattikar

Food Supplements | Food Additives |

Ayurveda and Vitamins | Nutraceuticals

Obesity and Ayurveda | Anorexia and Ayurveda

Immunity and Ayurveda | Immune Deficiency and Ayurveda

Exercise and Ayurveda | Muscle and Ayurveda | Fat and Ayurveda

Ayurvedic Yoga Therapy

Sex and Ayurveda | AIDS and Ayurveda | STDs and Ayurveda

Erectile Dysfunction and Ayurveda | Low Libido and Ayurveda

Ayurvedic Psychology | Sattva Rasas Tamas

Ayurvedic Meditation Guidelines | Vipassana and Ayurveda

Anxiety and Ayurveda | Anger and Ayurveda | Depression and Ayurveda

ADD and Ayurveda | Manic-Depression and Ayurveda | Overeating and Ayurveda

Technology and Ayurveda

Ayurvedic Mantras | Ayurvedic Yoga

Ayurvedic Spirituality | Nagarjuna Bodhisattva | Medicine Buddha

Medicine King Bodhisattva | Ksitigarbha Bodhisattva - Great Vows

Avalokiteshvara BodhisattvaGreat Compassion | Tara | Great Compassion Mantra

Manjushri Bodhisattva - Great Wisdom | Samantabhadra Bodhisattva - Great Practice

Maitreya Bodhisattva - Great Patience | Vajrapani Bodhisattva - Great Protection

Cause and Effect in Ayurveda | Dependent Origination | Ten Perfections

Five Precepts | Bodhisattva Precepts - Benefit Others More than Your Self

Ayurveda and the Bodhisattva Way

Ayurvedic Treatments | Ayurvedic Diseases | Ayurvedic Pathology

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Heart Disease and Ayurveda | Cancer and Ayurveda | Stroke and Ayurveda

Asthma and Ayurveda | Diabetes and Ayurveda | Alzheimers and Ayurveda

Arthritis and Ayurveda | Chronic Fatigue and Ayurveda

Insomnia and Ayurveda | Stress and Ayurveda

Pancha Karma | Ayurvedic Detox | Nasya Therapy | Donating Blood

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Ayurvedic ChromotherapyColor Therapy and Ayurveda

Ayurvedic Womans Health

Irregular Menstruation and Ayurveda | Painful Menstruation and Ayurveda

Yeast Infections and Ayurveda | Menopause and Ayurveda

Pregnancy and Ayurveda | Ayurvedic OBGYN |

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Visit our Kindred Sister Sites: (Different Non-Profit 501©3 Religious Organizations, but Shared Buddhist Ayurveda Values)

Medicine Buddha Healing Center and Ayurveda Healing Arts Institute:

Vajra Yogini Acupuncture Center and Earth Treasury Ayurveda Center:

Medicine Buddha Mantra: Om Namo Bhagavate Bhaisajya Guru Vaidurya Prabaha Rajaya Tathagataya Arhate Samyamsambodhi Tadyata Om Bhaisajye Bhaisajye Bhaisajya Samudgate Svaha!

Medicine King Bodhisattva Jeweled Ax Mantra 16 (Line 64 of the Great Compassion Mantra of Avalokiteshvara): Syi lu seng e mu chywe ye Nan Wei la ye Wei la ye Sa wa he.

osteoarthritis.txt · Last modified: 2018/02/26 18:12 (external edit)